The Continuation of the Virus – No Virus Discussion

Recently a few articles came out questioning the Viruses don’t exist arguments. One of them is by Doctors for Covid Ethics, written by Michael Palmer MD and Sukharit Bhakdi MD here:

They write that the baby has been thrown out with the bath water and indeed, some misguided zealots now claim germs don’t exist at all which is absurd. Bacteria, Parasites and Fungi have been clearly identified microscopically and proven to cause disease by truly isolating them and then following the Koch postulates. Anybody who ever took probiotic supplements will know that bacteria are real. We have billions of them living symbiotically in and on our bodies.  Claiming these germs don’t exist is (purposefully?) shutting down the discussion about the existence of viruses and making it appear blatantly ludicrous.

I also want to make clear that I have great respect for Dr. Sukharit Bhakdi. He has shown tremendous courage coming out in the early days after the roll out of the fake vaccines warning about their dangers. In no way do I want to diminish his contribution during the plandemic.

The article criticizes the stance that pure isolation of virus particles is necessary to begin proving their existence. It lists the following reasons:

The particles of many viruses have very characteristic shapes that are not likely to be confused with any particles produced by living cells, or with debris left behind by dead cells.
There are many biochemical methods for characterizing viral particles, and moreover for establishing that they contain genetic information characteristic of the virus rather than the host cell culture.

Not all viruses can easily be grown in cell cultures. Those which cannot are indeed routinely propagated in, and directly isolated from, laboratory animals.

A good example of such an animal study was published by Theil et al. It concerned the isolation of a novel virus from gnotobiotic, i.e. otherwise germ-free pigs.

The abstract reads:

A rotavirus-like virus (RVLV) was isolated from a diarrheic pig from an Ohio swine herd. This virus infected villous enterocytes throughout the small intestine of gnotobiotic pigs and induced an acute, transitory diarrhea. Complete virions [viral particles] were rarely observed in the intestinal contents of infected animals … The genome of the porcine RVLV was composed of 11 discrete segments of double-stranded RNA … that produced an electropherotype distinct from the genome electropherotypes of reovirus, rotavirus, and porcine pararotavirus. Porcine RVLV was antigenically unrelated to rotavirus, porcine pararotavirus, or reovirus but was antigenically related to a bovine RVLV.

The full PDF of the study can be downloaded here:

Let’s look at this study and ask some questions:

How was that rotavirus-like virus (RVLV) isolated from the pig?
According to the study they took intestinal contents of a pig with diarrhea. The study further describes that “The RVLV was initially detected, along with a rotavirus, in the intestinal contents of a gnotobiotic pig orally inoculated with a bacteria-free filtrate of the original specimen”.

Unfortunately, they don’t mention how RVLV or a rotavirus was “detected” in the inoculated pig. The study describes that they did genome electropherotyping to find rotavirus. Now, how do they know what the genome of a rotavirus is if that was done in the same way as with all the other “viruses” that were not properly isolated and mixed in with cells, bovine serum albumin and cells of the diseased animal and then compared to genetic sequences of other “viruses” not isolated in the same way?

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